The cells which have the capacity to multiply through out their life_____________?

Related Mcqs:

1. Why fetal cells continue to divide but terminally differentiated adult cells do not divide ?

   A. There are many cyclin inhibitors which prevent cell to enter into S phase in adult
   B. Phosphatase absent in fetal cells
   C. Proteinase is absent in fetus
   D. Absence of CD kinase

2. Bone marrow stem cells differ from differ from differentiated progenitor stem cells in what respect ?

   A. Provide differentiated terminal cells
   B. Reconstitution of Bone marrow
   C. Formation of the ovum
   D. Act as a repair system for the body

3. NK cells are effective against viral infected cells only if the cell with infection___________?

   A. Exprss MHC class I proteins
   B. Unable to express MHC class I proteins
   C. Express MHC class II proteins
   D. Unable to express MHC class II proteins

4. Vital capacity is decreased, timed vital capacity (FEV 1.0 %) is normal in______________?

   A. Bronchial asthma
   B. Scoliosis
   C. Chronic bronchitis
   D. Acute bronchitis

5. The cells which do not undergo mitotic divisions___________?

   A. Smooth muscle cells
   B. Endotheilial cells
   C. Bone marrow cells
   D. Neurons

6. All cells do not divide at the same rate. Events in which phase of the cell cycle determine when a cell is going to replicate ?

   A. M phase
   B. G1 phase
   C. S phase
   D. G2 phase

7. The persistence of epithelial cells after wound healing give rise to_______________?

   A. Implantation cyst
   B. Keloid
   C. Wound dehiscence
   D. Cicatrisation

8. Which of the following has least capacity for regeneration_________________?

   A. Cardiac muscle
   B. Skeletal muscle
   C. Neurons
   D. All of the above

9. In megaloblastic anaemia the cells are___________________?

   A. Macrocytic hyperchromic
   B. Macrocytic hypochromic
   C. Macrocytic normochromic
   D. None of the above

10. In acute inflammation, immediate transient permeability in cells is increased by__________?

    A. Direct injury & necrosis
    B. Endothelial Gaps by histamine
    C. Leucocyte mediated endothelial injury
    D. Increased tissue oncotic pressure